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It mainly acts on the N-methyl-D-aspartate (NMDA) receptor to produce sedative, analgesic, and anesthetic effects [8,9]. S(+)-ketamine is an effective S-enantiomer of racemic ketamine with a greater affinity than ketamine (approximately 2- to 4-fold) for NMDA receptors, thereby producing stronger sedative and analgesic effects and fewer adverse events, and it has been used to prevent central sensitization [10-13]. Opioids, such as remifentanil, may lead to hyperalgesia and more analgesic requirements.
JMIR Res Protoc 2023;12:e53063
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Opioid analgesic therapy, however, is only one part of a comprehensive multimodal pain treatment plan, which must also be covered in a comprehensive curriculum as well.
JMIR Med Educ 2023;9:e46784
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